The Human Liver: A Scanning Electron Microscopic Atlas. By F.‐J. Vonnahme. Basel: Karger, 1993. 100 pp. $65.75
Raymond S. Koff – 1 August 1994
Immunohistochemical phenotyping of liver macrophages in normal and diseased human liver
Minoru Tomita, Kazuhide Yamamoto, Haruhiko Kobashi, Masaki Ohmoto, Takao Tsuji – 1 August 1994 – The phenotypical heterogeneity of human liver macrophages was analyzed with monoclonal antibodies that recognize antigens specific for the monocytemacrophage lineage. Most liver macrophages in normal and diseased liver were positive for CD68, whereas fewer matured macrophages were detected by 25–F9. Comparative staining of mirror sections revealed some to be doubly positive and others to be singly CD68 positive.
Increased messenger RNA levels for low‐density lipoprotein receptor and 3‐hydroxy‐3‐methylglutaryl coenzyme a reductase in rat liver after long‐term ethanol ingestion
Helmut K. Seitz, Bernhard Kuhn, Eberhard von Hodenberg, Walter Fiehn, Christian Conradt, Ulrich A.
A 20‐year‐old woman with portal hypertension and a cholestatic syndrome
Leonardo Bianchi, Jürg Reichen – 1 August 1994
Biliary sludge: Curiosity or culprit?
Sum P. Lee, Alex Hayashi, Young S. Kim – 1 August 1994
Renal vasoconstriction in cirrhosis
Agostino Colli, Massimo Cocciolo, David Sacerdoti, Massimo Bolognesi, Carlo Merkel, Paolo Angeli, Angelo Gatta – 1 August 1994
Notice
1 August 1994
Renal duplex doppler ultrasonography: A noninvasive predictor of kidney dysfunction and hepatorenal failure in liver disease
Joel F. Platt, James H. Ellis, Jonathan M. Rubin, Robert M. Merion, Michael R. Lucey – 1 August 1994 – Hepatorenal failure, a well‐recognized complication of established liver disease, is characterized by early renal hemodynamic changes (vasoconstriction) before clinically recognized kidney disease. This renal vasoconstruction (increased renal vascular resistance) should be detectable noninvasively by Doppler ultrasonography.
AT long last: An animal model of Wilson's disease
Ronald J. Sokol – 1 August 1994 – The LEC rat is an inbred mutant strain with spontaneous hepatitis isolated from Long‐Evans rats. Since approximately 40% of LEC rats die of fulminant hepatitis, the rat serves an animal model for studying the pathogenesis and treatment of human fulminant hepatitis. The remaining 60% of LEC rats survive and develop chronic (prolonged) hepatitis and subsequently develop liver cancer.