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15 September 2018
Hepatology Highlights
Russell Rosenblatt, Nicholas Russo, Robert S. Brown, Nicole T. Shen, Robert E. Schwartz, Rachel Niec, Shawn Shah, Joseph F. Pisa, Vikas Gupta, Ashley Wu, Zaid H. Tafesh – 15 September 2018
Liver Transplantation for Fulminant Hepatitis Attributed to Yellow Fever
Alice Tung Wan Song, Edson Abdala, Rodrigo Bronze de Martino, Luis Marcelo Sá Malbouisson, Ryan Yukimatsu Tanigawa, Guilherme Marques Andrade, Liliana Ducatti, Andre Mario Doi, João Renato Rebello Pinho, Michele Soares Gomes‐Gouvêa, Fernanda de Mello Malta, Rubens Macedo Arantes, Adriana Coracini Tonacio, Lécio Figueira Pinto, Luciana Bertocco Paiva Haddad, Vinicius Rocha Santos, Rafael Soares Nunes Pinheiro, Lucas Souto Nacif, Flávio Henrique Ferreira Galvão, Venancio Avancini Ferreira Alves, Wellington Andraus, Luiz Augusto Carneiro D’Albuquerque – 15 September 2018
Hepatocyte‐Specific β‐Catenin Deletion During Severe Liver Injury Provokes Cholangiocytes to Differentiate Into Hepatocytes
Jacquelyn O. Russell, Wei‐Yu Lu, Hirohisa Okabe, Marc Abrams, Michael Oertel, Minakshi Poddar, Sucha Singh, Stuart J. Forbes, Satdarshan P. Monga – 14 September 2018 – Liver regeneration after injury is normally mediated by proliferation of hepatocytes, although recent studies have suggested biliary epithelial cells (BECs) can differentiate into hepatocytes during severe liver injury when hepatocyte proliferation is impaired. We investigated the effect of hepatocyte‐specific β‐catenin deletion in recovery from severe liver injury and BEC‐to‐hepatocyte differentiation.
The Interstitial Space Takes Shape
Prodromos Hytiroglou, Ian R. Wanless – 14 September 2018
Letter To Editor: Terlipressin is Superior to Noradrenaline in the Management of Acute Kidney Injury in Acute On Chronic Liver Failure
Sapana Verma, Shaleen Agarwal, Shweta Singh, Nivedita Pandey, Subhash Gupta – 14 September 2018
Inflammation and Microbiota Fingerprint: Delphi’s Oracle for Nonalcoholic Fatty Liver Disease–Related Hepatocellular Carcinoma?
Marco Cassano, Jean‐François Dufour – 14 September 2018
Hepatocyte‐Specific β‐Catenin Deletion During Severe Liver Injury Provokes Cholangiocytes to Differentiate Into Hepatocytes
Jacquelyn O. Russell, Wei‐Yu Lu, Hirohisa Okabe, Marc Abrams, Michael Oertel, Minakshi Poddar, Sucha Singh, Stuart J. Forbes, Satdarshan P. Monga – 14 September 2018 – Liver regeneration after injury is normally mediated by proliferation of hepatocytes, although recent studies have suggested biliary epithelial cells (BECs) can differentiate into hepatocytes during severe liver injury when hepatocyte proliferation is impaired. We investigated the effect of hepatocyte‐specific β‐catenin deletion in recovery from severe liver injury and BEC‐to‐hepatocyte differentiation.
De Novo Malignancies Screening After Liver Transplantation for Alcoholic Liver Disease: A Comparative Opportunistic Study
Laurence Renaud, Marie‐Noëlle Hilleret, Elsa Thimonier, Olivier Guillaud, Francois Arbib, Gilbert Ferretti, Adrien Jankowski, Christine Chambon‐Augoyard, Domitille Erard‐Poinsot, Thomas Decaens, Olivier Boillot, Vincent Leroy, Jérôme Dumortier – 12 September 2018 – Patients having received a liver transplantation (LT) for alcoholic liver disease (ALD) have a high risk of de novo malignancies, especially in the upper aerodigestive tract and lungs due to their smoking and alcohol history.
Changes in Cerebral Hemodynamics in Patients With Cirrhosis After Liver Transplantation
Ricardo Ulises Macías‐Rodríguez, Astrid Ruiz‐Margáin, Carlos Cantú‐Brito, Daniel Fernando Flores‐Silva, Octavio René García‐Flores, Francisco Javier Cubero, Elena Larrieta‐Carrasco, Aldo Torre – 12 September 2018 – Improvement in cognitive function after orthotopic liver transplantation (LT) has been demonstrated in the acute setting immediately after LT and in acute liver failure. However, the longterm changes in cerebral hemodynamics after LT remain unexplored. Therefore, we aimed to evaluate the longterm changes in cerebral hemodynamics of patients with cirrhosis after LT.