Hepatology Highlights
Russell Rosenblatt, Nicholas Russo, Robert S. Brown, Nicole T. Shen, Robert E. Schwartz, Rachel Niec, Shawn Shah, Joseph F. Pisa, Vikas Gupta, Ashley Wu, Zaid H. Tafesh – 15 September 2018
Russell Rosenblatt, Nicholas Russo, Robert S. Brown, Nicole T. Shen, Robert E. Schwartz, Rachel Niec, Shawn Shah, Joseph F. Pisa, Vikas Gupta, Ashley Wu, Zaid H. Tafesh – 15 September 2018
15 September 2018
15 September 2018
15 September 2018
15 September 2018
Jacquelyn O. Russell, Wei‐Yu Lu, Hirohisa Okabe, Marc Abrams, Michael Oertel, Minakshi Poddar, Sucha Singh, Stuart J. Forbes, Satdarshan P. Monga – 14 September 2018 – Liver regeneration after injury is normally mediated by proliferation of hepatocytes, although recent studies have suggested biliary epithelial cells (BECs) can differentiate into hepatocytes during severe liver injury when hepatocyte proliferation is impaired. We investigated the effect of hepatocyte‐specific β‐catenin deletion in recovery from severe liver injury and BEC‐to‐hepatocyte differentiation.
Marco Cassano, Jean‐François Dufour – 14 September 2018
Jacquelyn O. Russell, Wei‐Yu Lu, Hirohisa Okabe, Marc Abrams, Michael Oertel, Minakshi Poddar, Sucha Singh, Stuart J. Forbes, Satdarshan P. Monga – 14 September 2018 – Liver regeneration after injury is normally mediated by proliferation of hepatocytes, although recent studies have suggested biliary epithelial cells (BECs) can differentiate into hepatocytes during severe liver injury when hepatocyte proliferation is impaired. We investigated the effect of hepatocyte‐specific β‐catenin deletion in recovery from severe liver injury and BEC‐to‐hepatocyte differentiation.
Prodromos Hytiroglou, Ian R. Wanless – 14 September 2018
Sapana Verma, Shaleen Agarwal, Shweta Singh, Nivedita Pandey, Subhash Gupta – 14 September 2018