Notices
30 December 2003
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Thomas F. Imperiale, Naga Chalasani – 30 December 2003
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Josep M. Llovet, Ramon Vilana, Concepció Brú, Lluís Bianchi, Jordi Bruix – 30 December 2003
Tumor dissemination after radiofrequency ablation of hepatocellular carcinoma
Luigi Bolondi, Stefano Gaiani, Natascia Celli, Fabio Piscaglia – 30 December 2003
A confluence of epidemics: Does hepatitis C cause type 2 diabetes?
James Everhart – 30 December 2003
The ACAT2 gene encodes a gatekeeper of intestinal cholesterol absorption that regulates cholesterolemia and gallstone disease
Silvana Zanlungo, Flavio Nervi – 30 December 2003
Lowered nuclear levels of p53 in preneoplastic rat hepatocytes
Johan Högberg, Ulla Stenius – 30 December 2003
Prevalence and clinical course of chronic hepatitis C virus (HCV) infection and rate of HCV vertical transmission in a cohort of 15,250 pregnant women
Dario Conte, Mirella Fraquelli, Daniele Prati, Alice Colucci, Eliseo Minola – 30 December 2003 – The prevalence and natural course of chronic hepatitis C virus (HCV) infection was evaluated in 15,250 consecutive pregnant women. The rate of HCV vertical and perinatal transmission was also assessed. The presence of anti‐HCV was tested by means of EIA III and confirmed by recombinant immunoblot assay III.
Hepatitis C epidemiology: Injecting new tools in the field
David L. Thomas – 30 December 2003
Novel molecular defects of the δ‐aminolevulinate dehydratase gene in a patient with inherited acute hepatic porphyria
Reiko Akagi, Ryo Shimizu, Kazumichi Furuyama, Manfred O. Doss, Shigeru Sassa – 30 December 2003 – Cloning and expression of the defective gene for δ‐aminolevulinate dehydratase (ALAD) from the second of 2 German patients with ALAD deficiency porphyria (ADP), who had been originally reported by Doss et al. in 1979, were performed. Cloning of cDNAs for the defective ALAD were performed using Epstein‐Barr virus (EBV)‐transformed lymphoblastoid cells of the proband, and nucleotide sequences of cloned cDNA were determined. Two separate mutations of ALAD cDNA were identified in each ALAD allele.