Gastrointestinal emergencies. Edited by M.B. Taylor, 643 pp. Baltimore: Williams & Wilkins, 1992, $105
Daniel S. Camara – 1 July 1992
Daniel S. Camara – 1 July 1992
Shigeru Yotsumoto, Mineo Kojima, Isao Shoji, Kayoko Yamamoto, Hiroaki Okamoto, Shunji Mishiro – 1 July 1992 – A precore defective variant of hepatitis B virus has been indicated to cause fulminant hepatitis in various instances such as intrahospital outbreaks or motherto‐child transmission of hepatitis B virus.
Gilles A. Morali, Sheldon W. Tobe, Karl L. Skorecki, Laurence M. Blendis – 1 July 1992 – We have previously shown that unresponsiveness to atrial natriuretic factor is a marker of the severity of ascites. The tubular mechanisms are unknown, but it seems that increased reabsorption of sodium proximal to the main site of action of atrial natriuretic factor (i.e., the inner medullary collecting duct) plays an important role. We attempted to decrease the proximal reabsorption of sodium with mannitol in patients unresponsive to atrial natriuretic factor.
Dieter H. Meyer, Nora Krull, Kevin L. Dreher, Axel M. Gressner – 1 July 1992 – The expression of genes encoding the core proteins of the novel small chondroitin/dermatan sulfate proteoglycans decorin and biglycan was studied in the livers of normal rats and in liver tissue during fibrogenesis induced by prolonged bile‐duct ligation and thioacetamide poisoning. The cell types responsible for the expression of these transcripts and some key regulatory factors were identified. Both biglycan and decorin messenger RNAs were detected in normal liver tissue.
Carol Ann Toth, Dr.Peter Thomas – 1 July 1992
Dale L. Coy, Phyllis Zee, Andres T. Blei – 1 July 1992
Frieder Berr, Edgar Schreiber, Ulrich Frick – 1 July 1992 – The relative amount of cholesterol and the fatty acid composition of phosphatidylcholines in bile can be influenced by the bile acid species secreted.
A. William Paulsen, Goran B. G. Klintmalm – 1 July 1992 – The purpose of this study was to investigate intraoperatively a population of patients with end‐stage liver disease before and after liver transplantation with respect to (a) the range of hepatic and systemic hemodynamics and their changes associated with transplantation and (b) the ability to identify native hemodynamic correlates with specific diagnostic groups. Hepatic artery and portal vein blood flows were determined with square‐wave electromagnetic flowmetry.
Ikuko Motohashi, Masahiko Okudaira, Tomoko Takai, Satoshi Kaneko, Noriaki Ikeda – 1 July 1992 – In an attempt to provide a quantitative basis for differentiation between well‐differentiated hepatocellular carcinoma and hepatocellular carcinomalike lesions (focal nodular hyperplasia, regenerative nodular hyperplasia and hepatocellular adenoma), histopathological and morphometrical analyses were performed on 208 cases of various liver diseases with the aid of an image analyzer.