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Distinct and Common Fibrotic Pathways in Metabolic Dysfunction-Associated Steatohepatitis Versus MetALD

Description

Speakers present the common and distinct pathways involved in hepatic stellate cell activation and fibrogenesis in metabolic dysfunction-associated steatohepatitis (MASH) and MetALD in relation to disease-specific therapeutic approaches. 

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Objectives

  • Explain what is known about the common and distinct pathways contributing to fibrogenesis in MASH versus MetALD.
  • Describe how knowledge about the common and distinct pathways contributing to fibrogenesis in MASH and MetALD may aid in the design of disease-specific therapeutic interventions.